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Frontiers | Circadian Rhythm Dysfunction Accelerates Disease Progression in  a Mouse Model With Amyotrophic Lateral Sclerosis
Frontiers | Circadian Rhythm Dysfunction Accelerates Disease Progression in a Mouse Model With Amyotrophic Lateral Sclerosis

IJMS | Free Full-Text | Brain Protease Activated Receptor 1 Pathway: A  Therapeutic Target in the Superoxide Dismutase 1 (SOD1) Mouse Model of Amyotrophic  Lateral Sclerosis
IJMS | Free Full-Text | Brain Protease Activated Receptor 1 Pathway: A Therapeutic Target in the Superoxide Dismutase 1 (SOD1) Mouse Model of Amyotrophic Lateral Sclerosis

Amyotrophic lateral sclerosis (ALS):SOD1 mouse helps researchers identify  immune component
Amyotrophic lateral sclerosis (ALS):SOD1 mouse helps researchers identify immune component

Boosting the peripheral immune response in the skeletal muscles improved  motor function in ALS transgenic mice - ScienceDirect
Boosting the peripheral immune response in the skeletal muscles improved motor function in ALS transgenic mice - ScienceDirect

Zuoshang Xu Lab - University of Massachusetts Chan Medical School
Zuoshang Xu Lab - University of Massachusetts Chan Medical School

Gut microbes tune inflammation and lifespan in a mouse model of amyotrophic  lateral sclerosis
Gut microbes tune inflammation and lifespan in a mouse model of amyotrophic lateral sclerosis

Human iPSC-derived neural progenitor cells secreting GDNF provide  protection in rodent models of ALS and retinal degeneration - ScienceDirect
Human iPSC-derived neural progenitor cells secreting GDNF provide protection in rodent models of ALS and retinal degeneration - ScienceDirect

A major step towards the cure of sporadic ALS | The University of Tokyo
A major step towards the cure of sporadic ALS | The University of Tokyo

Working with ALS Mice: Guidelines for Preclinical Testing and Colony  Management
Working with ALS Mice: Guidelines for Preclinical Testing and Colony Management

IJMS | Free Full-Text | Novel P2X7 Antagonist Ameliorates the Early Phase  of ALS Disease and Decreases Inflammation and Autophagy in SOD1-G93A Mouse  Model
IJMS | Free Full-Text | Novel P2X7 Antagonist Ameliorates the Early Phase of ALS Disease and Decreases Inflammation and Autophagy in SOD1-G93A Mouse Model

From Mouse Models to Human Disease: An Approach for Amyotrophic Lateral  Sclerosis | In Vivo
From Mouse Models to Human Disease: An Approach for Amyotrophic Lateral Sclerosis | In Vivo

Opinion: more mouse models and more translation needed for ALS | Molecular  Neurodegeneration | Full Text
Opinion: more mouse models and more translation needed for ALS | Molecular Neurodegeneration | Full Text

AAV9-mediated gene delivery of MCT1 to oligodendrocytes does not provide a  therapeutic benefit in a mouse model of ALS: Molecular Therapy - Methods &  Clinical Development
AAV9-mediated gene delivery of MCT1 to oligodendrocytes does not provide a therapeutic benefit in a mouse model of ALS: Molecular Therapy - Methods & Clinical Development

Cortical Circuit Dysfunction as a Potential Driver of Amyotrophic Lateral  Sclerosis
Cortical Circuit Dysfunction as a Potential Driver of Amyotrophic Lateral Sclerosis

Murine Models of Neurodegenerative Diseases - Maze Engineers
Murine Models of Neurodegenerative Diseases - Maze Engineers

Molecules | Free Full-Text | A Novel Anti-Inflammatory d-Peptide Inhibits  Disease Phenotype Progression in an ALS Mouse Model
Molecules | Free Full-Text | A Novel Anti-Inflammatory d-Peptide Inhibits Disease Phenotype Progression in an ALS Mouse Model

Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic  lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular  atrophy protein to nuclear Cajal bodies. | Semantic Scholar
Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar

Humanising mice to enable modelling of neurodegenerative diseases | UCL  Queen Square Institute of Neurology - UCL – University College London
Humanising mice to enable modelling of neurodegenerative diseases | UCL Queen Square Institute of Neurology - UCL – University College London

November 2015 Pathomechanistic model mice of sporadic ALS: Conditional  ADAR2 knockout mice AR2 | Experimental Animal Division (RIKEN BRC)
November 2015 Pathomechanistic model mice of sporadic ALS: Conditional ADAR2 knockout mice AR2 | Experimental Animal Division (RIKEN BRC)

Amyotrophic Lateral Sclerosis: Marking the differences in motoneurons |  eLife
Amyotrophic Lateral Sclerosis: Marking the differences in motoneurons | eLife

Frontiers | Mouse Models of C9orf72 Hexanucleotide Repeat Expansion in Amyotrophic  Lateral Sclerosis/ Frontotemporal Dementia
Frontiers | Mouse Models of C9orf72 Hexanucleotide Repeat Expansion in Amyotrophic Lateral Sclerosis/ Frontotemporal Dementia

Rodent Amyotrophic Lateral Sclerosis (ALS) Model - Creative Biolabs
Rodent Amyotrophic Lateral Sclerosis (ALS) Model - Creative Biolabs

Figure 3.1 from The neuromuscular transmission of the SOD 1 ( G 93 A ) mouse  model of Amyotrophic Lateral Sclerosis | Semantic Scholar
Figure 3.1 from The neuromuscular transmission of the SOD 1 ( G 93 A ) mouse model of Amyotrophic Lateral Sclerosis | Semantic Scholar

Tempol improves neuroinflammation and delays motor dysfunction in a mouse  model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text
Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text

File:ALS animal models.jpg - Wikimedia Commons
File:ALS animal models.jpg - Wikimedia Commons

Laboratory Models of ALS - The ALS Association
Laboratory Models of ALS - The ALS Association

Modelling amyotrophic lateral sclerosis in rodents | Nature Reviews  Neuroscience
Modelling amyotrophic lateral sclerosis in rodents | Nature Reviews Neuroscience